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Rabbit Anti-KLRF2/Cy5.5 Conjugated antibody (bs-16785R-Cy5.5)
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說 明 書: 100ul  
100ul/2980.00元
大包裝/詢價
產(chǎn)品編號 bs-16785R-Cy5.5
英文名稱1 Rabbit Anti-KLRF2/Cy5.5 Conjugated antibody
中文名稱 Cy5.5標(biāo)記的殺傷細(xì)胞凝集素樣受體F2抗體
別    名 Activating coreceptor NKp65; Killer cell lectin-like receptor subfamily F member 2; KLRF2; KLRF2_HUMAN; Lectin-like receptor F2; NKp65.  
規(guī)格價格 100ul/2980元 購買        大包裝/詢價
說 明 書 100ul  
研究領(lǐng)域 細(xì)胞生物  免疫學(xué)  自然殺傷細(xì)胞  
抗體來源 Rabbit
克隆類型 Polyclonal
交叉反應(yīng) Human, 
產(chǎn)品應(yīng)用 ICC=1:50-200 IF=1:50-200 
not yet tested in other applications.
optimal dilutions/concentrations should be determined by the end user.
分 子 量 24kDa
性    狀 Lyophilized or Liquid
濃    度 1mg/ml
免 疫 原 KLH conjugated synthetic peptide derived from human KLRF2
亞    型 IgG
純化方法 affinity purified by Protein A
儲 存 液 0.01M TBS(pH7.4) with 1% BSA, 0.03% Proclin300 and 50% Glycerol.
保存條件 Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.
產(chǎn)品介紹 background:
C-type lectin-like receptor involved in natural killer cell mediated cytotoxicity and cytokine secretion in keratinocytes via its interaction with CLEC2A.

Function:
C-type lectin-like receptor involved in natural killer cell mediated cytotoxicity and cytokine secretion in keratinocytes via its interaction with CLEC2A.

Subunit:
Homodimer; non disulfide-linked. Interacts with CLEC2A.

Subcellular Location:
Cell membrane.

Tissue Specificity:
Contains 1 C-type lectin domain.

Post-translational modifications:
N-glycosylated.

Similarity:
Contains 1 C-type lectin domain.

Database links:

Entrez Gene: 100431172 Human

SwissProt: D3W0D1 Human

Unigene: 725113 Human



Important Note:
This product as supplied is intended for research use only, not for use in human, therapeutic or diagnostic applications.
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